RBM4B Rabbit Polyclonal Antibody, 50ul Protein Expression Expression of a kinase-defective mutant

Expression of a kinase-defective mutant results in abnormal cell morphology caused by changes in microtubule dynamics and mitotic arrest followed by apoptosis

eceptor for N-arachidonyl glycine

It is expressed in the central and peripheral nervous system where it may be involved in increasing oxygen availability and providing protection under hypoxic/ischemic conditions

Alternative splicing and the use of multiple translation start codons results in multiple transcript variants encoding different isoforms

Activation of TLRs by PAMPs leads to an up-regulation of signaling pathways to modulate the host's inflammatory response

RBM4B Rabbit Polyclonal Antibody, 50ul Protein Expression Expression of a kinase-defective mutant