K-Ras Polyclonal Antibody, 20ul Peptide Modification may modulate vesicle budding and

may modulate vesicle budding and uncoating within the Golgi apparatus

This subunit may associate with a beta 1 or beta 4 subunit to form an integrin that interacts with extracellular matrix proteins including members of the laminin family

Don't let anything hold you back

Defects in this gene are a cause of juvenile-onset pulverulent cataract as well as congenital cerulean cataract 4 (CCA4)

which plays an important role in maintenance of muscle structure and function

K-Ras Polyclonal Antibody, 20ul Peptide Modification may modulate vesicle budding andKRAS (KRAS proto oncogene, GTPase), a Kirsten ras oncogene homolog from the mammalian ras gene family, encodes a protein that is a member of the small GTPase superfamily. A single amino acid substitution is responsible for an activating mutation. The transforming protein that results is implicated in various malignancies, including lung adenocarcinoma, mucinous adenoma, ductal carcinoma of the pancreas and colorectal carcinoma. Alternative splicing