MAGA6 Polyclonal Antibody, 20ul Cell Dissociation Defects in EP300 are a

Defects in EP300 are a cause of Rubinstein-Taybi syndrome and may also play a role in epithelial cancer

Mitochondrial ribosomes (mitoribosomes) consist of a small 28S subunit and a large 39S subunit

It contains an N-terminal caspase recruitment domain (CARD)| a centrally located nucleotide-binding domain (NBD)| and 10 tandem leucine-rich repeats (LRRs) in its C terminus

KRT10 encodes a member of the type I (acidic) cytokeratin family

This protein plays a role in maintaining cellular structural integrity and also functions in signal transduction and cellular differentiation

MAGA6 Polyclonal Antibody, 20ul Cell Dissociation Defects in EP300 are aThis gene is a member of the MAGEA gene family. The members of this family encode proteins with 50 to 80% sequence identity to each other. The promoters and first exons of the MAGEA genes show considerable variability, suggesting that the existence of this gene family enables the same function to be expressed under different transcriptional controls. The MAGEA genes are clustered at chromosomal location Xq28. They have been implicated in some