KPSH2 Polyclonal Antibody, 20ul Gene Editing Defects in ATG16L1 are a

Defects in ATG16L1 are a cause of susceptibility to inflammatory bowel disease type 10 (IBD10)

including AMP-morpholidate

GPR37 is a member of the G protein-coupled receptor family

Alternatively spliced transcript variants encoding distinct isoforms have been observed

ADCK2 participates in ATP and nucleotide binding

KPSH2 Polyclonal Antibody, 20ul Gene Editing Defects in ATG16L1 are aCatalytic activity: ATP + a protein = ADP + a phosphoprotein.,Asn 183 is present instead of the conserved Asp which is expected to be an active site residue.,Belongs to the protein kinase superfamily. CAMK Ser Thr protein kinase family.,Contains 1 protein kinase domain.,